Laszlo Markasz
Affiliated Researcher at Department of Women's and Children's Health; Perinatal, Neonatal and Pediatric Cardiology Research
- E-mail:
- laszlo.markasz@uu.se
- Visiting address:
- MTC-huset, Dag Hammarskjölds väg 14B, 1 tr
752 37 Uppsala - Postal address:
- Akademiska sjukhuset
751 85 UPPSALA - Available:
- Neonatalavdelningen 95F
- Academic merits:
- Associate Professor in Pediatrics, MD, PhD
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Publications
Recent publications
- Collagen type IV alpha 1 chain (COL4A1) expression in the developing human lung (2024)
- Paneth cell proteins DEFA6 and GUCA2A as tissue markers in necrotizing enterocolitis (2023)
- Vascular adhesion protein-1 expression is reduced in the intestines of infants with necrotizing enterocolitis (2022)
- Early Postnatal Comprehensive Biomarkers Cannot Identify Extremely Preterm Infants at Risk of Developing Necrotizing Enterocolitis. (2021)
- The value of autopsy in preterm infants at a Swedish tertiary neonatal intensive care unit 2002-2018 (2021)
All publications
Articles
- Collagen type IV alpha 1 chain (COL4A1) expression in the developing human lung (2024)
- Paneth cell proteins DEFA6 and GUCA2A as tissue markers in necrotizing enterocolitis (2023)
- Vascular adhesion protein-1 expression is reduced in the intestines of infants with necrotizing enterocolitis (2022)
- Early Postnatal Comprehensive Biomarkers Cannot Identify Extremely Preterm Infants at Risk of Developing Necrotizing Enterocolitis. (2021)
- The value of autopsy in preterm infants at a Swedish tertiary neonatal intensive care unit 2002-2018 (2021)
- CD44 and RHAMM expression patterns in the human developing lung (2021)
- Cluster Analysis of Early Postnatal Biochemical Markers May Predict Development of Retinopathy of Prematurity (2020)
- The receptor for hyaluronan-mediated motility (RHAMM) expression in neonatal bronchiolar epithelium correlates negatively with lung air content (2018)
- Diminished DEFA6 Expression in Paneth Cells Is Associated with Necrotizing Enterocolitis (2018)
- Inhibition of protein phosphatase-1 and -2A decreases the chemosensitivity of leukemic cells to chemotherapeutic drugs (2015)