Omar Hmeadi
Postdoktor vid Institutionen för medicinsk cellbiologi; Forskargrupp Sebastian Barg
- Telefon:
- 018-471 42 92
- E-post:
- Omar.Hmeadi@mcb.uu.se
- Besöksadress:
- BMC
Husargatan 3
751 23 Uppsala - Postadress:
- Box 571
751 23 UPPSALA
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Publikationer
Senaste publikationer
- Local PI(4,5)P2 signaling inhibits fusion pore expansion during exocytosis (2023)
- Functional, metabolic and transcriptional maturation of human pancreatic islets derived from stem cells. (2022)
- Alternative splicing encodes functional intracellular CD59 isoforms that mediate insulin secretion and are down-regulated in diabetic islets (2022)
- Insulin granule biogenesis and exocytosis (2021)
- Paracrine control of α-cell glucagon exocytosis is compromised in human type-2 diabetes. (2020)
Alla publikationer
Artiklar
- Local PI(4,5)P2 signaling inhibits fusion pore expansion during exocytosis (2023)
- Functional, metabolic and transcriptional maturation of human pancreatic islets derived from stem cells. (2022)
- Alternative splicing encodes functional intracellular CD59 isoforms that mediate insulin secretion and are down-regulated in diabetic islets (2022)
- Insulin granule biogenesis and exocytosis (2021)
- Paracrine control of α-cell glucagon exocytosis is compromised in human type-2 diabetes. (2020)
- Fusion pore regulation by cAMP/Epac2 controls cargo release during insulin exocytosis (2019)
- The Endoplasmic Reticulum Chaperone Glucose-Regulated Protein 94 is Essential for Proinsulin Handling (2019)
- Glucose-Dependent Granule Docking Limits Insulin Secretion and Is Decreased in Human Type 2 Diabetes (2018)
- PtdIns(4,5)P2 is not required for secretory granule docking (2018)
- Syntaxin clusters at secretory granules in a munc18-bound conformation (2018)
- Plasma membrane PI(4,5)P-2 is critical for secretory granule exocytosis (2017)
- Fusion pore regulation by transient local generation of PI(4,5)P2 in pancreatic in β-cells
- Paracrine control of α-cell glucagon exocytosis is compromised in human type-2 diabetes