Konstantin Gängel
- E-post:
- konstantin.gaengel@igp.uu.se
- Besöksadress:
- Dag Hammarskjölds väg 20
751 85 Uppsala - Postadress:
- Rudbecklaboratoriet
751 85 UPPSALA
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Publikationer
Senaste publikationer
- Incongruence between transcriptional and vascular pathophysiological cell states (2023)
- Cellular Origin of Sporadic CCMs (2022)
- Mural Cell SRF Controls Pericyte Migration, Vessel Patterning and Blood Flow (2022)
- Tamoxifen-independent recombination of reporter genes limits lineage tracing and mosaic analysis using CreER(T2) lines (2020)
- Sphingosine 1-Phosphate Receptor Signaling Establishes AP-1 Gradients to Allow for Retinal Endothelial Cell Specialization (2020)
Alla publikationer
Artiklar
- Incongruence between transcriptional and vascular pathophysiological cell states (2023)
- Cellular Origin of Sporadic CCMs (2022)
- Mural Cell SRF Controls Pericyte Migration, Vessel Patterning and Blood Flow (2022)
- Tamoxifen-independent recombination of reporter genes limits lineage tracing and mosaic analysis using CreER(T2) lines (2020)
- Sphingosine 1-Phosphate Receptor Signaling Establishes AP-1 Gradients to Allow for Retinal Endothelial Cell Specialization (2020)
- CDC42 deletion elicits cerebral vascular malformations via increased MEKK3-dependent KLF4 expression (2019)
- Integrins are required for synchronous ommatidial rotation in the Drosophila eye linking planar cell polarity signalling to the extracellular matrix (2019)
- Prickle is phosphorylated by Nemo and targeted for degradation to maintain Prickle/Spiny-legs isoform balance during planar cell polarity establishment (2018)
- Defective endothelial cell migration in the absence of Cdc42 leads to capillary-venous malformations (2018)
- New imaging methods and tools to study vascular biology (2015)
- Gpr116 Receptor Regulates Distinctive Functions in Pneumocytes and Vascular Endothelium (2015)
- Endocytosis regulates VEGF signalling during angiogenesis (2013)
- Cdc42 is required in mural cells for proper patterning of the retinal vasculature
- Defective endothelial cell migration in the absence of Cdc42 leads to capillary-venous malformations